|NEWS AND FILLER
|Year : 2012 | Volume
| Issue : 1 | Page : 70-71
Dyselectrolytemias: Word search
A Gupta, M Biyani
Division of Nephrology, University of Ottawa, Ottawa, Canada
|Date of Web Publication||26-Dec-2011|
Unit 318, 1833 Riverside Drive, Ottawa, ON, K1G 0E8
Source of Support: None, Conflict of Interest: None
|How to cite this article:|
Gupta A, Biyani M. Dyselectrolytemias: Word search. Indian J Nephrol 2012;22:70-1
| Clues|| |
- Proton pump inhibitors and epidermal growth factor receptor antagonists may cause this dyselectrolytemia.
- Polyuria, hypokalemia, and hypomagnesemia occurring in this infective cause of acute kidney injury.
- Cyclical excessive vomiting and compulsive taking of hot baths with this substance abuse.
- Syndrome characterized by hypokalemic alkalosis, hypomagnesemia, and hypocalciuria.
- This root extract may cause mineralocorticoid excess.
- Possible adverse effect of bevacizumab.
- Continuous renal replacement therapy causing low ionized calcium, high anion gap metabolic acidosis, and this toxicity in liver failure patients.
- Electrolyte imbalance may induce this syndrome characterized by ST segment elevation in right precordial leads followed by negative T wave unrelated to ischemia on electrocardiogram.
- Hyponatremia after the use of this alkylating agent.
- Syndrome characterized by skin hyperpigmentation, polyostotic fibrous dysplasia, and hypophosphatemia.
- Mutation in this gene along with elevated fibroblast growth factor (FGF)-23 causes X-linked hypophosphatemic rickets.
- FGF-23, FGF-7, matrix extracellular phosphoglycoprotein, and secreted frizzled-related protein-4 are examples of .......onin.
- Mutation in with-no-lysine kinases (WNK) 1 and 4 are responsible for this hyperkalemic syndrome.
- Aldosterone-sensitive Cl - /HCO3 - exchanger in cortical collecting duct encoded by Slc26a4.
- A cofactor mandatory for FGF-23 action.
- Spurious hyperphosphatemia after the use of this locking agent in hemodialysis.
- Imatinib causes renal wasting of this electrolyte.
- Cause of monogenic hypercalciuric nephrolithiasis.
- Autosomal recessive (AR) lysosomal storage disorder causing nephrogenic diabetes insipidus.
- Antibiotic causing neurotoxicity and not so commonly hyponatremia.
- Hyperchloremic metabolic acidosis and kidney stones with this antiseizure drug.
- Pseudo-Bartter syndrome developing in AR disorder of Cl - channel defects.
- Intestinal syndrome with hypokalemia.
- Vitamin A intoxication and this dyselectrolytemia.
- Angiotensin-converting enzyme inhibitors may cause this metabolic abnormality.
- Antibiotic class associated with hypokalemia and metabolic alkalosis.
- Dyselectrolytemia occurring by placing blood sample on ice and resultant urgent nephrology consult.
- Acute severe oleander poisoning causing this dyselectrolytemia.
- Be careful of potassium correction in this dyselectrolytemia.
- Agents to enhance free water excretion.
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