CASE REPORT |
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Year : 2016 | Volume
: 26
| Issue : 6 | Page : 461-463 |
Acquired Bartter syndrome following gentamicin therapy
J Singh1, ML Patel1, KK Gupta1, S Pandey2, A Dinkar3
1 Department of Medicine, King George Medical University, Lucknow, Uttar Pradesh, India 2 Department of Nephrology, King George Medical University, Lucknow, Uttar Pradesh, India 3 Department of Microbiology, Institute of Medical Science, BHU, Varanasi, Uttar Pradesh, India
Correspondence Address:
J Singh Department of Medicine, King George's Medical University, Lucknow, Uttar Pradesh India
 Source of Support: None, Conflict of Interest: None  | Check |
DOI: 10.4103/0971-4065.177206
Aminoglycoside nephrotoxicity may manifest as nonoliguric renal failure or tubular dysfunction, such as Fanconi-like syndrome, Bartter-like syndrome (BS), or distal renal tubular acidosis. We report a case who developed severe renal tubular dysfunction on the the 7 th day of gentamicin therapy, resulting in metabolic alkalosis, refractory hypokalemia, hypocalcemia, hypomagnesemia, and polyuria. The patient was diagnosed as a case of transient BS associated with gentamicin exposure. The patient recovered with conservative management.
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