Acquired Bartter syndrome following gentamicin therapy

J Singh; ML Patel; KK Gupta; S Pandey; A Dinkar

doi:10.4103/0971-4065.177206

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CASE REPORT

Year : 2016 | Volume : 26 | Issue : 6 | Page : 461-463

Acquired Bartter syndrome following gentamicin therapy

J Singh¹, ML Patel¹, KK Gupta¹, S Pandey², A Dinkar³
¹ Department of Medicine, King George Medical University, Lucknow, Uttar Pradesh, India
² Department of Nephrology, King George Medical University, Lucknow, Uttar Pradesh, India
³ Department of Microbiology, Institute of Medical Science, BHU, Varanasi, Uttar Pradesh, India

Correspondence Address:
J Singh
Department of Medicine, King George's Medical University, Lucknow, Uttar Pradesh
India

Source of Support: None, Conflict of Interest: None

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DOI: 10.4103/0971-4065.177206

Aminoglycoside nephrotoxicity may manifest as nonoliguric renal failure or tubular dysfunction, such as Fanconi-like syndrome, Bartter-like syndrome (BS), or distal renal tubular acidosis. We report a case who developed severe renal tubular dysfunction on the the 7 ^th day of gentamicin therapy, resulting in metabolic alkalosis, refractory hypokalemia, hypocalcemia, hypomagnesemia, and polyuria. The patient was diagnosed as a case of transient BS associated with gentamicin exposure. The patient recovered with conservative management.

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