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Year : 2016  |  Volume : 26  |  Issue : 2  |  Page : 149--150

Renal tuberculosis with lobar calcification

A Balani1, AK Dey2, SS Sarjare1, A Narkhede1,  
1 Department of Radiology, Yashoda Hospital, Secunderabad, Telangana, India
2 Department of Radiology, Seth GS Medical College and KEM Hospital, Mumbai, Maharashtra, India

Correspondence Address:
A K Dey
Department of Radiology, Seth GS Medical College and KEM Hospital, Room No. 107, Main Boy«SQ»s Hostel, Acharya Donde Marg, Parel, Mumbai - 400 012, Maharashtra

How to cite this article:
Balani A, Dey A K, Sarjare S S, Narkhede A. Renal tuberculosis with lobar calcification .Indian J Nephrol 2016;26:149-150

How to cite this URL:
Balani A, Dey A K, Sarjare S S, Narkhede A. Renal tuberculosis with lobar calcification . Indian J Nephrol [serial online] 2016 [cited 2021 Nov 30 ];26:149-150
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A 32-year-old male presented with right-sided abdominal pain. Physical examination revealed right-sided renal angle tenderness. The temperature was 38.7°C and blood pressure 170/82 mmHg. The hemogram and biochemistries were within the normal limits except for elevated serum creatinine levels (1.8 mg/dl). X-ray of the abdomen and pelvis showed small right kidney with pathognomonic lobar calcification, with calcific rims around the distorted lobes, along with ureteric calcification [Figure 1]a]. Computerized tomography (CT) confirmed the findings of lobar renal calcifications with ureteric calcification suggesting genito-urinary tuberculosis (TB) [Figure 1]b]. There was history of pulmonary TB at the age of 19, for which he had taken anti-tuberculous treatment for 6 months. The diagnosis of urinary TB was confirmed by urine culture. The patient was started on anti-tuberculous treatment and surgery was advised for the management of hypertension but the patient refused.{Figure 1}

Tuberculosis of the genito-urinary tract usually affects adults between 20 and 40 years of age and is rare in children. [1] Diagnosis is usually delayed because of insidious onset and nonspecific symptoms of presentation such as fever, weight loss, anorexia, abdominal pain, dysuria and sometimes hematuria. [1]

The causative organism is usually Mycobacterium tuberculosis, which disseminates hematogenously or lymphatically to the kidneys. [1],[2]

The pattern of presentation of calcification ranges from initial faint and punctate to later coalescing. Focal granulomatous mass is usually encountered with focal lobar calcifications. The occurrence of ureteral calcification along with renal calcifications further proves the diagnosis of lobar presentation and generally has unfavorable prognosis. [1],[3]

Intravenous urogram is regarded as the investigations of choice that provides with anatomical and functional details. Earliest findings include minimal calyceal dilatation with the loss of calyceal sharpness. Ultrasonography or CT plays an important role not only in evaluating nonfunctioning kidneys but also in patients with complications of renal TB such as perinephritis, perinephric abscess, fistulae, psoas abscesses and rarely renal failure. [1],[4]


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